I was speaking of the genetic makeup that one couldn’t possibly know.
Ie. is activating more androgen receptors going to be sub optimal for you because your myostatin kicks in and make gaining extra muscle/strength harder for you than most?
or
is the strength gain coming from hyperplasia going to be less, more, equal to what you get with greater androgen receptivity?
etc.
The kind of answers that they’ve have to cut off half your leg for science to get reliable results.
I don’t talk about much of my experience unless needed (and it’s not here)
I already said former myostatin fields have gotten me “big” without a regimented workout routine.
Don’t have St. Biceps to compare.
But I am the one who originally said
Take that for what it’s worth considering I’m not showing pics and not terribly interested in convincing anyone of this.
As said before… just trying to clear up some “bro-science(sis-science)” around these fields and how muscles “work” in general.